Molecular Mechanisms in the Pathogenesis of Idiopathic Nephrotic
Molecular Mechanisms in the Pathogenesis of Idiopathic Nephrotic SyndromeAuthor(s): Kazunari Kaneko\nFormat: Hardback\nPublisher: Springer Verlag, Japan, Japan\nImprint: Springer Verlag, Japan\nISBN-13: 9784431552697, 978-4431552697\nSynopsis\nThis comprehensive book reviews our current state of knowledge about the pathogenesis of idiopathic nephrotic syndrome (INS), which comprises a heterogeneous group of diseases with distinct histological characteristics, such as minimal-change nephrotic syndrome (MCNS), focal segmental glomerulosclerosis (FSGS), and idiopathic membranous nephropathy (IMN). As the word idiopathic indicates, the pathogenesis of INS remains unclear. Historically, T-cell dysfunction has been thought to play an important part in the pathogenesis of MCNS, while circulating vascular permeabilities have been believed to induce proteinuria in FSGS. The book further describes recent advances in molecular biology, which have allowed us to speculate on the interactions betw.
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